If you haven’t enabled third-party account restore in Duo Mobile then app backups to Google account backup (Android) or iCloud (iOS) accounts DO NOT contain any private key or other sensitive data. Duo Mobile’s restore functionality lets you back up Duo-protected accounts and third-party OTP accounts (such as Google or Facebook) for recovery to the same device or to a new device. We speculate that the expression of heterochronic genes controlling the L2/L3 programs, including that of hbl-1 and lin-42, are increased during L1 diapause to arrest the developmental progression, and miR-71 is probably required to suppress these “excess” signals during the recovery phase (Fig. S5). Furthermore, the observed derepression of individual genes by mir-71(lf) seemed too weak to account for the phenotype, consistent with the idea that a prominent phenotype of an miRNA mutation is caused by the collective effect of changing expression in many genes, an important property of miRNA-mediated gene regulation. (F) Fluorescence and DIC images showing that an hbl-1 3′UTR reporter was repressed in mir-71(+) worms and slightly derepressed in mir-71(lf) mutants.

The Recovery and Resilience Facility in your country

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• If you haven’t enabled third-party account restore in Duo Mobile then app backups to Google account backup (Android) or iCloud (iOS) accounts DO NOT contain any private key or other sensitive data.
• The two ain-1 loss-of-function alleles displayed significant reductions in L1 starvation survival rate.
• The Duo Mobile accounts list shows your restored Duo accounts, and you may use them to log into Duo-protected services with Duo Push or a generated passcode.
• The RRF Regulation requires that the Commission provides the European Parliament, the Council, the European Economic and Social Committee and the Committee of the Regions with a mid-term evaluation on the implementation of the Recovery and Resilience Facility.
• We then compared the expression of a hbl-1 3′UTR reporter (18) in the mir-71(lf) mutants with that in wild type and found that the expression of this reporter was slightly derepressed at L3 in the mir-71 mutant (Fig. 4 F and G).
• A total of 16–24 h later, the density of newly hatched L1 worms was adjusted to three to five worms per microliter S-basal.
• (C) The reduced L1 starvation survival rate of ain-1(lf) mutants was significantly suppressed by a null allele of unc-31.